지원사업
학술연구/단체지원/교육 등 연구자 활동을 지속하도록 DBpia가 지원하고 있어요.
커뮤니티
연구자들이 자신의 연구와 전문성을 널리 알리고, 새로운 협력의 기회를 만들 수 있는 네트워킹 공간이에요.
논문 기본 정보
- 자료유형
- 학술저널
- 저자정보
- 발행연도
- 2009.6
- 수록면
- 9 - 14 (6page)
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Chemotherapy induced cardiotoxicity is a well-known side effect of several cytotoxic drugs, especially of the anthracyclines and may cause severe morbidity in surviving cancer patients. The mechanism of anthracycline induced cardiotoxicity, relatively well established, seems to involve the free radicals formation and the apoptosis of myocytes. Prevention of cardiac toxicity can be achieved by control of the cumulative dose. Furthermore, addition of the iron chelator to anthracycline therapy has shown to be helpful. Other cytotoxic drugs such as the taxoids, 5-fluorouracil, cyclophosphamide and trastuzumab are associated with cardiotoxicity as well, although little is known about the possible mechanisms. The pathophysiology of these adverse effects is similarly heterogeneous and the identification of potential mechanisms is frequently difficult since the majority of cancer patients is not only treated with multiple anti-cancer drugs but might also be exposed to potentially cardiotoxic radiation therapy. Further research is warranted to understand the mechanism of chemotherapy induced cardiotoxicity and to develop strategies to avoid this side effect.
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UCI(KEPA) : I410-ECN-0101-2014-513-001300488