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논문 기본 정보
- 자료유형
- 학술대회자료
- 저자정보
- 발행연도
- 2021.7
- 수록면
- 203 - 203 (1page)
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Immune cells that promote inflammation are recruited to inflamed and damaged tissues through vessels. Helper T (TH) cell infiltration are also densely observed in inflamed region. Cytokines such as interleukin regulate the development of naive CD4+ T cells into TH1, TH2, TH17 or regulatory T (Treg) cells. Dysregulation of TH17 cells plays important roles in various autoimmune diseases, including rheumatoid arthritis (RA). However, crosstalk between T cells and endothelial cells is not fully understood; whether angiogenic factors influence the TH differentiation or whether a specific TH subset directly contributes to pathologic angiogenesis in autoimmune inflammatory diseases. Since CD4+ T cells are primarily infiltrated in the inflammatory region, antigen-and cytokine-activated T cells are can be exposed to a variety of angiogenic factors, including vascular endothelial growth factor (VEGF). These angiogenic factors ... 전체 초록 보기
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