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논문 기본 정보
- 자료유형
- 학술저널
- 저자정보
- 발행연도
- 2006.1
- 수록면
- 216 - 221 (6page)
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Cyclosporin A-induced gingival hyperplasia is frequently found in the patients who have been received an immunosuppressant for the organ transplantation.
However, its exact mechanism is still unknown. The expression of FGF-5 and FGF-7 were studied in cyclosporine A-induced gingival
hyperplasia (CGH) and inflammatory gingival hyperplasia (IGH). Immunohistochemistry and in situ hybridization were used for localization of protein
and mRNA. The expression of FGF-5 and FGF-7 was different from CGH and IGH. FGF-5 and FGF-7 was strongly expressed in fibroblast in
CGH (P<0.005 and P<0.05, respectively). FGF-5 mRNA was localized in the middle portion of connective tissue. FGF-7 mRNA was also identified
in fibroblasts and mast cells. In conclusion, FGF-5 and FGF-7 were produced excessively by fibroblasts in CGH. Considering their known functions,
their expression in CGH is important for production of collagen and proliferation of fibroblasts.
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참고문헌 (14)
참고문헌 신청
/ 1985 / The effects of cyclosporin A on the immune system 3 : 397 ~ 423
/ 1989 / Regulation of human gingival fibroblast growth and syntheticactivity by cyclosporin-A in vitro 24 : 314 ~ 321
/ 1990 / Mast cell are not involved in the development of cyclosporin A-induced gingival hyperplasia:A study with mast cell-deficient mice 71 : 1117 ~ 1120
/ 1995 / Cyclosporine stimulates expression oftransforming growth factor-beta in renal cells.Possible mechanism ofcyclosporines proliferative effects 60 : 237 ~ 241
/ 1995 / Fas ligation induces apoptosis of CD40-activatedhuman B lymphocytes 182 : 1265 ~ 1273
/ 1989 / Regulation of human gingival fibroblast growth and syntheticactivity by cyclosporin-A in vitro 24 : 314 ~ 321
/ 1990 / Mast cell are not involved in the development of cyclosporin A-induced gingival hyperplasia:A study with mast cell-deficient mice 71 : 1117 ~ 1120
/ 1995 / Cyclosporine stimulates expression oftransforming growth factor-beta in renal cells.Possible mechanism ofcyclosporines proliferative effects 60 : 237 ~ 241
/ 1995 / Fas ligation induces apoptosis of CD40-activatedhuman B lymphocytes 182 : 1265 ~ 1273
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