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논문 기본 정보
- 자료유형
- 학술저널
- 저자정보
- 발행연도
- 2021.1
- 수록면
- 12 - 28 (17page)
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Both acute and chronic kidney disease have a strong underlying inflammatory component. This review focuses primarily on T helper 17 (Th17) cells as mediators of inflammation and their potential to modulate acute and chronic kidney disease. We provide updated information on factors and signaling pathways that promote Th17 cell differentiation with specific reference to kidney disease. We highlight numerous clinical studies that have investigated Th17 cells in the setting of human kidney disease and provide updated summaries from various experimental animal models of kidney disease indicating an important role for Th17 cells in renal fibrosis and hypertension. We focus on the pleiotropic effects of Th17 cells in different renal cell types as potentially relevant to the pathogenesis of kidney disease. Finally, we highlight studies that present contrasting roles for Th17 cells in kidney disease progression.
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참고문헌 (147)
참고문헌 신청
Webster AC / 2017 / Chronic kidney disease / Lancet 389:1238~1252
Basile DP / 2012 / Pathophysiology of acute kidney injury / Compr Physiol 2:1303~1353
Chertow GM / 2006 / Mortality after acute renal failure : models for prognostic stratification and risk adjustment / Kidney Int 70:1120~1126
Chertow GM / 2005 / Acute kidney injury, mortality, length of stay, and costs in hospitalized patients / J Am Soc Nephrol 16:3365~3370
Susantitaphong P / 2013 / World incidence of AKI : a meta-analysis / Clin J Am Soc Nephrol 8:1482~1493
Basile DP / 2012 / Pathophysiology of acute kidney injury / Compr Physiol 2:1303~1353
Chertow GM / 2006 / Mortality after acute renal failure : models for prognostic stratification and risk adjustment / Kidney Int 70:1120~1126
Chertow GM / 2005 / Acute kidney injury, mortality, length of stay, and costs in hospitalized patients / J Am Soc Nephrol 16:3365~3370
Susantitaphong P / 2013 / World incidence of AKI : a meta-analysis / Clin J Am Soc Nephrol 8:1482~1493
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