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논문 기본 정보
- 자료유형
- 학술저널
- 저자정보
- 발행연도
- 2018.1
- 수록면
- 354 - 362 (9page)
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초록· 키워드
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Cellular stress severely disrupts endoplasmic reticulum (ER) function, leading to the abnormal accumulation of unfolded or misfolded proteins in the ER and subsequent development of endoplasmic reticulum stress (ERS). To accommodate the occurrence of ERS, cells have evolved a highly conserved, selfprotecting signal transduction pathway called the unfolded protein response. Notably, ERS signaling is involved in the development of a variety of diseases and is closely related to tumor development, particularly in breast cancer. This review discusses recent research regarding associations between ERS and tumor metastasis. The information presented here will help researchers elucidate the precise mechanisms underlying ERS-mediated tumor metastasis and provide new directions for tumor therapies.
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참고문헌 (91)
참고문헌 신청
DeSantis C / 2014 / Breast cancer statistics, 2013 / CA Cancer J Clin 64:52~62
Torre LA / 2015 / Global cancer statistics, 2012 / CA Cancer J Clin 65:87~108
Wang M / 2016 / Protein misfolding in the endoplasmic reticulum as a conduit to human disease / Nature 529:326~335
Yu H / 2011 / p62/SQSTM1 involved in cisplatin resistance in human ovarian cancer cells by clearing ubiquitinated proteins / Eur J Cancer 47:1585~1594
Li Y / 2014 / New insights into the roles of CHOP-induced apoptosis in ER stress / Acta Biochim Biophys Sin(Shanghai) 46:629~640
Torre LA / 2015 / Global cancer statistics, 2012 / CA Cancer J Clin 65:87~108
Wang M / 2016 / Protein misfolding in the endoplasmic reticulum as a conduit to human disease / Nature 529:326~335
Yu H / 2011 / p62/SQSTM1 involved in cisplatin resistance in human ovarian cancer cells by clearing ubiquitinated proteins / Eur J Cancer 47:1585~1594
Li Y / 2014 / New insights into the roles of CHOP-induced apoptosis in ER stress / Acta Biochim Biophys Sin(Shanghai) 46:629~640
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