지원사업
학술연구/단체지원/교육 등 연구자 활동을 지속하도록 DBpia가 지원하고 있어요.
커뮤니티
연구자들이 자신의 연구와 전문성을 널리 알리고, 새로운 협력의 기회를 만들 수 있는 네트워킹 공간이에요.
논문 기본 정보
- 자료유형
- 학술저널
- 저자정보
- 발행연도
- 2016.1
- 수록면
- 265 - 275 (11page)
이용수
초록· 키워드
오류제보하기
Hemodynamic management of sepsis-induced circulatory failure is complex since this pathological state includes multiple cardiovascular derangements that can vary from patient to patient according to the degree of hypovolemia, of vascular tone depression, of myocardial depression and of microvascular dysfunction. The treatment of the sepsis-induced circulatory failure is thus not univocal and should be adapted on an individual basis. As physical examination is insufficient to obtain a comprehensive picture of the hemodynamic status, numerous hemodynamic variables more or less invasively collected, have been proposed to well assess the severity of each component of the circulatory failure and to monitor the response to therapy. In this article, we first describe the hemodynamic variables, which are the most relevant to be used, emphasizing on their physiological meaning, their validation and their limitations in patients with septic shock. We then proposed a general approach for managing patients with septic shock by describing the logical steps that need to be followed in order to select and deliver the most appropriate therapies. This therapeutic approach is essentially based on knowledge of physiology, of pathophysiology of sepsis, and of published data from clinical studies that addressed the issue of hemodynamic management of septic shock.
목차
등록된 정보가 없습니다.
참고문헌 (43)
참고문헌 신청
Walley KR / 1996 / Heterogeneity of oxygen delivery impairs oxygen extraction by peripheral tissues: theory / J Appl Physiol (1985) 81:885~894
Kreymann G / 1993 / Oxygen consumption and resting metabolic rate in sepsis, sepsis syndrome, and septic shock / Crit Care Med 21:1012~1019
Levy B / 2008 / Increased aerobic glycolysis through beta2 stimulation is a common mechanism involved in lactate formation during shock states / Shock 30:417~421
Levy B / 2005 / Relation between muscle Na+K+ ATPase activity and raised lactate concentrations in septic shock: a prospective study / Lancet 365:871~875
Levraut J / 1998 / Mild hyperlactatemia in stable septic patients is due to impaired lactate clearance rather than overproduction / Am J Respir Crit Care Med 157(4 Pt 1):1021~1026
Kreymann G / 1993 / Oxygen consumption and resting metabolic rate in sepsis, sepsis syndrome, and septic shock / Crit Care Med 21:1012~1019
Levy B / 2008 / Increased aerobic glycolysis through beta2 stimulation is a common mechanism involved in lactate formation during shock states / Shock 30:417~421
Levy B / 2005 / Relation between muscle Na+K+ ATPase activity and raised lactate concentrations in septic shock: a prospective study / Lancet 365:871~875
Levraut J / 1998 / Mild hyperlactatemia in stable septic patients is due to impaired lactate clearance rather than overproduction / Am J Respir Crit Care Med 157(4 Pt 1):1021~1026
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