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학술연구/단체지원/교육 등 연구자 활동을 지속하도록 DBpia가 지원하고 있어요.
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연구자들이 자신의 연구와 전문성을 널리 알리고, 새로운 협력의 기회를 만들 수 있는 네트워킹 공간이에요.
논문 기본 정보
- 자료유형
- 학술저널
- 저자정보
- 발행연도
- 2018.1
- 수록면
- 1 - 15 (15page)
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The term “vulnerable plaque” denotes the plaque characteristics that are susceptible to coronary thrombosis. Previous post-mortem studies proposed 3 major mechanisms of coronary thrombosis: plaque rupture, plaque erosion, and calcified nodules. Of those, characteristics of rupture-prone plaque have been extensively studied. Pathology studies have identified the features of rupture-prone plaque including thin fibrous cap, large necrotic core, expansive vessel remodeling, inflammation, and neovascularization. Intravascular imaging modalities have emerged as adjunctive tools of angiography to identify vulnerable plaques. Multiple devices have been introduced to catheterization laboratories to date, including intravascular ultrasound (IVUS), virtual-histology IVUS, optical coherence tomography (OCT), coronary angioscopy, and near-infrared spectroscopy. With the use of these modalities, our understanding of vulnerable plaque has rapidly grown over the past several decades. One of the goals of intravascular imaging is to better predict and prevent future coronary events, for which prospective observational data is still lacking. OCT delineates microstructures of plaques, whereas IVUS visualizes macroscopic vascular structures. Specifically, plaque erosion, which has been underestimated in clinical practice, is gaining an interest due to the potential of OCT to make an in vivo diagnosis. Another potential future avenue for intravascular imaging is its use to guide treatment. Feasibility of tailored therapy for acute coronary syndromes (ACS) guided by OCT is under investigation. If it is proven to be effective, it may potentially lead to major shift in the management of millions of patients with ACS every year
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참고문헌 (68)
참고문헌 신청
Falk E / 1983 / Plaque rupture with severe pre-existing stenosis precipitating coronary thrombosis. Characteristics of coronary atherosclerotic plaques underlying fatal occlusive thrombi / Br Heart J 50:127~134
Friedman M / 1966 / Role of thrombus in plaque formation in the human diseased coronary artery / Br J Exp Pathol 47:550~557
Willerson JT / 1984 / Conversion from chronic to acute coronary artery disease: speculation regarding mechanisms / Am J Cardiol 54:1349~1354
Davies MJ / 1990 / A macro and micro view of coronary vascular insult in ischemic heart disease / Circulation 82:II38~II46
Davies MJ / 1985 / Plaque fissuring--the cause of acute myocardial infarction, sudden ischaemic death, and crescendo angina / Br Heart J 53:363~373
Friedman M / 1966 / Role of thrombus in plaque formation in the human diseased coronary artery / Br J Exp Pathol 47:550~557
Willerson JT / 1984 / Conversion from chronic to acute coronary artery disease: speculation regarding mechanisms / Am J Cardiol 54:1349~1354
Davies MJ / 1990 / A macro and micro view of coronary vascular insult in ischemic heart disease / Circulation 82:II38~II46
Davies MJ / 1985 / Plaque fissuring--the cause of acute myocardial infarction, sudden ischaemic death, and crescendo angina / Br Heart J 53:363~373
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